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What this means is that treating prostate and breasts cancers require completely opposing approaches to AR. In treating prostate cancers, the strategy should be to block AR; in breasts cancer, the strategy ought to be to support AR production. Experts from Cleveland Clinic’s Lerner Analysis Institute, including Charis Eng, M.D., Ph.D., Chair, Genomic Medicine Institute; Robert Silverman, Ph.D., and Warren Heston, Ph.D., both of the Department of Cancer Biology; focused on if the androgen receptor molecule offers proof the tumor suppressor protein PTEN. The extensive research found that AR inhibits PTEN expression in prostate cancers cells, but stimulates it in breasts cancer cells.